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Arch Derm Syphilol. Sep;56(3) A case for diagnosis: lichen ruber moniliformis (?), dermatitis actinica from roentgen ray therapy of legs. OBERMAYER. Dermatitis Actinica; Dr. Selden I. Rainforth (American, – ); ; Color Photomechanical; XC; J. Paul Getty Museum, Los Angeles. Dermatologic stereoview card. Front of card depicts a man with Dermatitis Actinica on his face. The reverse of the card describes the dermatologic conditions as.

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The reverse of the card describes the dermatologic conditions as well as the diagnosis and treatment. Imiquimod is a topical immune-enhancing agent licensed for the treatment of genital warts. Unsourced or poorly sourced material may be challenged and removed.

The New England Journal of Medicine. International Journal of Dermatology. Patients of either sex, presenting to our department, clinically suggestive of having chronic actinic dermatitis were enrolled.

Orphanet: Dermatite actinica cronica

Please help improve this article by adding citations to reliable sources. Extremely cautious desensitizing with photochemotherapy PUVA or narrowband UVB with systemic steroid cover has been successful in some cases. Skin nearby the lesion often shows evidence of solar damage characterized by notable pigmentary alterations, being yellow or pale in color with areas of hyperpigmentation; deep wrinkles, coarse texture, purpura and acfinicadry skinand scattered telangiectasias are also characteristic.

Actinic keratosis and squamous cell carcinoma SCC can present similarly on physical exam, and many scientists argue that they are in fact simply different stages actinlca the same condition. Therapeutics and Clinical Risk Management.

Epidermal nevus Syndromes Epidermal nevus syndrome Schimmelpenning syndrome Nevus comedonicus syndrome Nevus comedonicus Inflammatory linear verrucous epidermal nevus Linear verrucous epidermal nevus Pigmented hairy epidermal nevus syndrome Systematized epidermal nevus Phakomatosis pigmentokeratotica. Several predictors for increased AK risk from UV radiation have been identified:.


Actinic reticuloid syndrome is considered the most severe stage of chronic actinic dermatitis CAD. Cure rates from 67 to 99 percent dermaitis been reported, [74] [75] depending on freeze time and lesion characteristics. Men are more likely to develop AK than women, and the risk of developing AK lesions increases with age.

Harry Moseley 25 Estimated H-index: The reactions that develop confirm the presence of an abnormal reaction to the light. A medium-depth peel may effectively treat multiple non-hyperkeratotic AKs. Left untreated, there is a chance that the lesion will advance to become invasive. Critical review and meta-analysis of efficacy studies”.

Dermtitis lo considera en el espectro de las dermatitis actinicas cronicas DAC el polo mas severo.

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A chronic red or eczematous rash, usually on the face or exposed skin surfaces, that typically results from exposure and sensitization to ultraviolet rays. However a biopsy may be necessary when achinica keratosis is large in diameter, thick, or bleeding, in order to make sure that the lesion is not a skin cancer.

YAG lasers is a treatment approach being utilized with increased frequency, and sometimes in conjunction with computer scanning technology.

Chronic actinic dermatitis secondary to simvastatin. Aggressive digital papillary adenocarcinoma Extramammary Paget’s disease.

Chronic actinic dermatitis CAD is an immunologically mediated photodermatosis usually observed in temperate climates and that typically develops in middle-aged to elderly males. This article needs additional citations for verification. In terms of treatment, a number of medications are being studied. A case of false-negative monochromator phototesting in a patient with chronic actinic dermatitis taking prednisolone. The seven major histopathologic variants are all characterized by atypical keratinocytic proliferation beginning in the basal layer and confined to the epidermis; they include: Lasers in Surgery and Medicine.


It is thought that ultraviolet UV radiation induces mutations in the keratinocytes of the epidermis, promoting the survival and proliferation of these atypical cells. If a shave biopsy is performed, it should extend through to the level of the dermis in order to provide sufficient tissue for diagnosis; ideally, it would extend to the mid-reticular dermis. Ultraviolet radiation is believed to contribute to the development of actinic keratoses by inducing mutations in epidermal keratinocytes, leading to proliferation of atypical cells.

Upregulation of matrix metalloproteinases MMP is seen in many different types of cancers, and the expression and production of MMP-7 in particular has been found to be elevated in SCC specifically. This involves specialized tests in which areas of skin are exposed to known amounts of light of specific wavelengths. For most people it is a lifelong condition that requires significant lifestyle changes to avoid sunlight as well as contact allergens.

If you have any concerns with your skin or its treatment, see a dermatologist for advice. The most important cause of AK formation is solar radiation, through a variety of mechanisms.